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However, these strains show more severe hemorrhage caused by sinusoidal endothelial cell (SEC) damage [99, 129], which may aggravate the hepatocellular injury due to microcirculatory disturbances leading to additional ischemic necrosis [95, 129, 130]

The abnormal proliferation of specific microbial taxa and their metabolites (such as SCFAs and tryptophan derivatives) contribute to SLE development by compromising intestinal barrier integrity, triggering autoimmune responses, and promoting systemic inflammation