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The metabolic effects of AKI within the kidney are well-described: disruption of mitochondrial structure, dynamics, biogenesis, and basic electron transport chain function are all present 36

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Immunofluorescence staining showed that, compared with IL-1-treated ECs, ferrostatin-1-induced inhibition of ferroptosis restored ZO-1 and connexin 43 fluorescence intensity in IL-1-treated ECs (Fig

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