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Therefore, it is urgent to study the molecular mechanisms of ferroptosis in the progression of aortic dissection and to explore treatment strategies for delaying its progression

Extracellular N-terminal hydrophobic domains, tryptophan residues22), alanine residues23), and cysteine residues24) are critical for the interaction between GLP-1 and GLP-1R

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Monomeric compounds such as Astragaloside IV and Quercetin precisely intervene in the mTOR/NF-B/NLRP3 axis, while compound preparations such as Lingguizhugan Decoction and Qishen Yiqi Formula strengthen the regulation of immune-metabolic networks through the synergistic effect of multiple components, providing new strategies for the development of natural drugs for PH treatment