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Cela permet une absorption optimale des nutriments

For instance, KRAS mutations drive cancer cell metabolic dependency by increasing glycolysis and subsequently feeding the ser/gly synthesis side branch via NRF2 upregulation, which is a transcription factor that is frequently deregulated in NSCLC [6]

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Tailoring treatments according to the distinct features of the breast tumor microenvironment and its interactions with different molecular subtypes of breast cancer may facilitate the development of personalized medicine approaches