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doi: 10.1007/s00432-024-05917-w

In sequence, -synuclein can trigger NLRP3 inflammasome with subsequent release of IL-1 with the development of systemic inflammation and neuroinflammation (Zhang et al

OSU-T315: a novel targeted therapeutic that antagonizes AKT membrane localization and activation of chronic lymphocytic leukemia cells

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GHK-Cu treatment reduced reactive oxygen species (ROS) production, increased superoxide dismutase (SOD) activity, and decreased TNF- and IL-6 production by inhibiting NF-B p65 and p38 MAPK signaling in LPS-induced macrophages and ALI models