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Recent studies have revealed an autoregulatory mechanism whereby mitochondrial GSH (mtGSH) homeostasis is controlled through a feedback loop involving solute carrier family 25 member 39 (SLC25A39) transporter and mitochondrial protease AFG3 like matrix AAA peptidase subunit 2 (AFG3L2), coupling GSH levels to iron-sulfur cluster biogenesis 16,17
Treatment with this conjugate resulted in strong CD8 + T cells-mediated cytotoxicity against MUC1-positive tumor cells, inhibiting tumor growth and prolonging survival [207]
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