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The mechanisms underlying these potential synergistic effects remain under active investigation and may involve growth factor signaling, extracellular matrix synthesis, or anti-catabolic pathways in joint tissues

Patients with pre-existing severe motility issues may experience worse symptoms

5 Circadian rhythms regulate post-IS BBB repair via the neuroendocrine system 5.1 The neuroendocrine-circadian axis The neuroendocrine system acts as a key interface linking circadian regulation to BBB homeostasis and contributes to barrier repair after ischemic stroke

A previous study demonstrated that HFD-fed, STZ-induced, insulin-resistant diabetic mice are good models for patients with T2DM that have risk factors for obesity and distinct metabolic characteristics (35)