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Inducible nitric oxide synthase gene deficiency counteracts multiple manifestations of peripheral neuropathy in a streptozotocin-induced mouse model of diabetes
These peptide hormones were developed initially as a type 2 diabetes treatment that works with the pancreas to enhance insulin release in response to meals
However, joints are also sites where oxidative stress can accumulate, especially after intense physical activity
If ERS persists and does not alleviate, UPR will induce apoptosis
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