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By stimulating collagen synthesis and reorganizing damaged tissue fibers, the skin begins to appear plumper, more supple, and younger over time

Its working mechanism is allosteric facilitation of HGF/c-Met signaling: Dihexa binds HGF with high affinity (reported K d ~65 pM in the original work) and is proposed to potentiate HGF dimerization and c-Met phosphorylation at subthreshold endogenous HGF concentrations

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So what is it exactly

One of the fundamental reflections that emerge from our experience is that the correct timing for reducing therapy must be identified according to the patient, who has to be correctly informed about the symptoms that may appear in this process